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In addition, OXM treatment improved glucose tolerance in high-fat fed mice by potentiating GSIS (111), in a glucose-dependent manner (112), and has an anti-apoptotic effect on cells (112)

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The same cellular mechanisms that help reduce stubborn fatnamely, the inhibition of the NNMT enzyme and the resulting preservation of NAD+also play a fundamental role in how our bodies age and manage energy system-wide